Gout is a type of arthritis that occurs when too much uric acid builds up. It causes glass like crystals to form in joints, aggravates the kidneys (acid urine and stone formation) and potentiates high blood pressure and cardiovascular disease.
Hyperuricemia (gout) is diagnosed with a blood test of serum uric acid (SUA) above 420 μM (70 μg/ml).
It can be hereditary in nature, or the result of another condition. Gout usually affects men over forty with a family history of gout. Certain foods, alcohol, surgery, infection, physical or emotional stress, various drugs can contribute to the development of gout symptoms.
extreme pain in a single joint, usually the base of the big toe, but can also affect the feet, fingers, wrists, elbows, knees or ankles
joint becomes shiny red, purple, swollen, hot and stiff
fever high as 39 °C (102.2 °F), could have chills
rapid onset, usually comes at night, relieves in 5-10 days, with potential to return.
Acid, burning urine, cramping (stone formation); at a urine pH of 5.3, 50% of uric acid will be stone forming
Symptoms of a kidney stone mimic appendicitis. Signs to watch for are nausea, vomiting, blood in the urine, sudden, severe pain that gets worse in waves. Stones may cause intense pain in the back, side, abdomen, groin, or genitals.
As disease progress, may see uric acid crystals come to surface; lumps under skin at outer ear, hands, feet, elbow, knee
Eliminating all forms of alcohol is often all it takes to prevent gouty arthritis in many people. Alcohol increases uric acid production and reduces uric acid excretion. As alcohol is digested it breaks down accelerates purine nucleotide production (increases uric acid). Drinking or storing alcohol from leaded crystal may contribute to the build of uric acid because the lead consumed will impair the kidney’s ability to eliminate uric acid.
How diet can help
Purines are a type of protein that gets metabolized into uric acid. A low-purine diet has long been known to alleviate the symptoms of gout. However, with the advent of potent drugs that lower uric acid levels, many physicians lower the serum urate levels without the inconvenience and deprivation associated with a purine-free diet. Furthermore, there are naturopathic remedies to help ease the strain of hyperuricemia. Regardless, dietary restriction of purines is recommended to reduce metabolic stress.
General diet guidelines:
Eliminate alcohol intake
Low purine intake
Achievement of ideal body weight
Liberal consumption of complex carbohydrates
Low fat intake
Low protein intake
6-8 glasses pure water per day. More if working in a active environment or where you may sweat more (increased loss of fluids)
Organ meats, meats, pork, gravy
Fish such as salmon, perch, pickerel, tilapia,
Protein < 0.8g/kg of body weight
Shellfish, herring, sardines, mackerel, and anchovies
Low fat milk and yogurt
Cherries, blueberries, blackberries
Yeast (brewer’s and baker’s) watch bread consumption.
Refined carbohydrates (i.e. breads, cakes, cookies, pasta) as they increase uric acid
Unleavened breads, squash, sweet potato
Niacin (>50mg/day) Competes for uric acid excretion Mega doses of vitamin C as may increase uric acid levels
Limit foods containing oxalate, such as spinach, rhubarb, beets, nuts, chocolate, black tea, wheat bran, strawberries, and beans.
Lots of green leafy vegetables and cauliflower, kale broccoli & cabbage
Alcohol of any kind
Sugar sweetened soft drinks.
6-8 glasses pure water per day
Saturated fats (increase uric acid retention)
Use coconut or olive oil for cooking
*Nightshades (tomatoes, potatoes, bell peppers, eggplant) can sometimes affect those with a family history of rheumatoid arthritis.
Naturopathic Treatment Options
Naturopathic medicine is powerful and unique to the individual and design to remove obstacles to healing and support that body’s natural mechanisms of health. Types of therapies vary and can include:
botanical (plant based medicine)
Choi HK. A prescription for lifestyle change in patients with hyperuricemia and gout. [Review]. Curr Opin Rheumatol. 2010;22(2):165-72.
Choi HK. Diet, alcohol, and gout: how do we advise patients given recent developments? Curr RheumatolRep. 2005;7(3):220-6.
Choi HK, Curhan G. Coffee consumption and risk of incident gout in women: the Nurses’ Health Study. Am J Clin Nutr. 2010;92(4):922-7.
Choi HK, Curhan G. Soft drinks, fructose consumption, and the risk of gout in men: prospective cohort study. BMJ. 2008; [Epub ahead of print].
Choi HK, Gao X, Curhan G. Vitamin C intake and the risk of gout in men: a prospective study. Arch Intern Med. 2009;169(5):502-7.
Dubchak N, Falasca GF. New and improved strategies for the treatment of gout. Int J Nephrol Renovasc Dis. 2010;3:145-66.
Eggebeen AT. Gout: an update. Am Fam Physician. 2007;76(6):801-8. Review.
Li S, Micheletti R. Role of diet in rheumatic disease. [Review]. Rheum Dis Clin North Am. 2011;37(1):119-33.
Pascual E, Sivera F. Therapeutic advances in gout. Curr Opin Rheumatol. 2007;19(2):122-7.
Peterson DM. Nonsteroidal anti-inflammatory drugs and colchicine to prevent gout flare during early urate-lowering therapy: perspectives on alternative therapies and costs. J Pain Palliat Care Pharmacother. 2010;24(4):402-4.
Pizzorno J., Murray M., Textbook of Natural Medicine 3rd ed. Seattle. Churchill Livingston Elselvier. 2005.
Richette P, Bardin T. Gout. Lancet. 2010;375(9711):318-28.
Saag KG, Choi H. Epidemiology, risk factors, and lifestyle modifications for gout. ArthritisRes Ther. 2006;8 Suppl 1:S2.
Schelesinger N. Overview of the management of acute gout and the role of adrenocorticotropic hormone. Drugs. 2008; 68(4):407-15.
Schlesinger N, Dalbeth N, Perez-Ruiz F. Gout — what are the treatment options? Expert Opin Pharmacother. 2009;10(8):1319-28.
Schumacher HR Jr, Chen LX. Newer therapeutic approaches: gout. Rheum Dis Clin North Am. 2006;32(1):235-44, xii. Review.
Suresh E, Das P. Recent advances in management of gout. QJM. 2011 Dec 23. [Epub ahead of print].
Vaghamshi R, Jaiswal M, Patgiri BJ, Prajapati PK, Ravishankar B, Shukla VJ. A comparative pharmacological evaluation of Taila (oil) and Ghrita (ghee) prepared with Guduchi (Tinospora cordifolia). Ayu. 2010;31(4):504-8.
Wegener T, Lupke NP. Treatment of patients with arthrosis of hip or knee with an aqueous extract of devil’s claw (Harpagophytum procumbens DC.). Phytother Res 2003;17(10):1165-1172.
Wiederkehr, M. R., & Moe, O. W. (2011). Uric Acid Nephrolithiasis: A Systemic Metabolic Disorder. Clinical reviews in bone and mineral metabolism, 9(3-4), 207–217. https://doi.org/10.1007/s12018-011-9106-6
Zhang SJ, Liu JP, He KQ. Treatment of acute gouty arthritis by blood-letting cupping plus herbal medicine. J Tradit Chin Med. 2010;30(1):18-20.